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Table 1 Postemergence maturation defects associated with disabling RK function in different cell types

From: The tanning hormone, bursicon, does not act directly on the epidermis to tan the Drosophila exoskeleton

Tissue targeted1Driver2Pigmentation defects3Sclerotization defects4Wing expansion defects5Cuticle appearance6
RK cellsrk-GAL4+++ 7+++++++++
EpidermisTH-GAL4+++
Tendon cellssr-GAL4
Hemocyteshem-GAL4
MusclesC59-GAL4
Oenocytesdesat(RE)-GAL4
Neuronselav-GAL4++++++++++++
nsyb-GAL4+++ND++++++
Peptidergic neuronsamon-GAL4+++ND+++++
dimm-gal4++ND
ILP7 neuronsilp7-GAl4++ND
  1. 1Tissues in which RK function was disabled by driving tBur
  2. 2GAL4 driver used to drive UAS-tBur in desired tissues
  3. 3–6 Defects observed in pigmentation 3; sclerotization 4 (assessed measuring soluble protein present in cuticle); in wing expansion 5 and in cuticle appearance 6 (incomplete maturation causes cuticle to have a surface that is matte in appearance)
  4. 7 Defects were classified qualitatively as severe (+++; similar to those of rk mutant animals) to normal (−). ND not determined