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Fig. 6 | BMC Biology

Fig. 6

From: A primary cell wall cellulose-dependent defense mechanism against vascular pathogens revealed by time-resolved dual transcriptomics

Fig. 6

Upregulation of ET signaling contributes to the resistance of cellulose-deficient mutants during Fo5176 infection. A Root vascular penetration of the ET signaling mutant ein2-5 in WT (Col-0) and ctl1-2 genetic backgrounds at various days post-treatment (dpt) with Fo5176 pSIX1::GFP microconidia. Values represent the mean ± standard error of at least 3 independent experiments, each one containing at least 16 seedlings. Statistical significance calculated via repeated measures two-way ANOVA with Tukey post-hoc test (p value ≤ 0.05 (genotype), p value ≤ 0.05 (time), p value ≤ 0.05 (genotype x time)). Significant differences at the last time point shown (7 dpt) are indicated on the graph using letters; statistics of remaining time points summarized in Additional file 9: Table S4A-F. B Representative images of 8-day-old light-grown seedlings impaired in ET signaling in WT (Col-0) or ctl1-2 background. C Quantification of root length of plants grown as depicted in E. Bars represent the mean ± standard deviation of N ≥ 40 plants averaged over three independent experiments. Statistical significance calculated via one-way ANOVA with Tukey post-hoc test (p value < 0.05). Significant differences indicated using letters

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